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ÀÇ´ë º»°ú 4Çгâ PK °úÁ¦(°ú¹Î¼º ÆóÀå¿°)(2)

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2018.05.08
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9 page / 53.6 KB
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inflammation of the alveoli within the lung caused by hypersensitivity to inhaled organic dusts.sufferers are commonly exposed to the dust by their occupation or hobbies.
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inhalation of an antigen.
this leads to an exaggerated immune response (hypersensitivity).
type iii hypersensitivity and type iv hypersensitivity occur in hypersensitivity pneumonitis
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1)thermophilic actinomycetes:most common(°í¿Â ¹æ»ç¼±±Õ)
2)±âŸ Áø±Õ,µ¿½Ä¹° ºÐºñ¹°.¿øÃæ.È­Çй°Áú, ¾à¹° µî
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5)Èí¿¬ÀÚ´Â ±Þ¼ºº¸´Ù ¸¸¼ºÀÇ ÇüÅ°¡ ¸¹´Ù
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symptoms may develop xxx-xxx hours following heavy exposure to the provoking antigen.
fever, chills, malaise, cough, chest tightness, dyspnea, and headache.symptoms resolve within 12 hours to several days upon cessation of exposure.
poorly formed noncaseating interstitial granulomas and mononuclear cell infiltration in a peribronchial distribution with prominent giant cells.
chest radiographs: a diffuse micronodular interstitial pattern (at times with ground-glass density in the lower and middle lung zones)
in high-resolution ct scans: ground-glass opacities or diffusely increased radiodensities
pulmonary function tests : reduced diffusion capacity of lungs for carbon monoxide (dlco).
-hypoxemia at rest, and all patients desaturate with exercise.
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acute    
productive cough, dyspnea, fatigue, anorexia, weight loss, and pleurisy.
less severe and last longer.
on chest radiographs, micronodular or reticular opacities are most prominent in mid-to-lower lung zones
noncaseating granulomas, bronchiolitis with or without organizing pneumonia, and interstitial fibrosis.
subacute       (ÀÌÇÏ »ý·«)

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